Friday, December 12, 2008
Friday, November 28, 2008
Inverse Organs
My heart is on the right side, and all my organs are switch, left right, and right left...
I am Diagnostic with kartagener's syndrome with Chronic Bronchitis and Bronchiectasis.
Select and Read about each case under the Diagnostic Posts Category.
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I am Diagnostic with kartagener's syndrome with Chronic Bronchitis and Bronchiectasis.
Select and Read about each case under the Diagnostic Posts Category.
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Saturday, October 18, 2008
kartagener's Syndrome:
What is Kartagener's Syndrome?
Kartagener's Syndrome is a chronic lung disease that occurs when cilia are unable to move. Kartagener's Syndrome is a rare genetic birth defect.
Individuals with Kartagener's Syndrome have abnormal or absent ciliary motion.
Kartagener's Syndrome is also called Primary Ciliary Dyskinesia, and Immotile Cilia Syndrome, Afzelius’ syndrome, Kartagener’s triad, Zivert's syndrome, and Zivert-Kartagener triad.
What are Cilia?
Cilia are tiny hair-like structures that are present on the surfaces of many different types of cells.
One of the functions of cilia is to move mucus out of the respiratory passages.
During infections more mucus is produced and because the abnormal cilia can't move or move ineffectively, mucus becomes stuck and blocks the respiratory tract, causing various complications.
Complications of Kartagener's Syndrome
Some complications of Kartagener's Syndrome are:
Bronchiectasis.
Bronchiectasis occurs when the bronchial tubes are blocked and chronically infected, causing the tubes to weaken and stretch out.
The widened tubes allow more mucus and bacteria to accumulate, leading to infection and possibly pneumonia.
Difficulty breathing.
Chronic wheezing, coughing.
Kartagener's Syndrome is a chronic lung disease that occurs when cilia are unable to move. Kartagener's Syndrome is a rare genetic birth defect.
Individuals with Kartagener's Syndrome have abnormal or absent ciliary motion.
Kartagener's Syndrome is also called Primary Ciliary Dyskinesia, and Immotile Cilia Syndrome, Afzelius’ syndrome, Kartagener’s triad, Zivert's syndrome, and Zivert-Kartagener triad.
What are Cilia?
Cilia are tiny hair-like structures that are present on the surfaces of many different types of cells.
One of the functions of cilia is to move mucus out of the respiratory passages.
During infections more mucus is produced and because the abnormal cilia can't move or move ineffectively, mucus becomes stuck and blocks the respiratory tract, causing various complications.
Complications of Kartagener's Syndrome
Some complications of Kartagener's Syndrome are:
Bronchiectasis.
Bronchiectasis occurs when the bronchial tubes are blocked and chronically infected, causing the tubes to weaken and stretch out.
The widened tubes allow more mucus and bacteria to accumulate, leading to infection and possibly pneumonia.
Difficulty breathing.
Chronic wheezing, coughing.
Chronic Bronchitis:
Bronchitis is an inflammation of the bronchi.
Symptoms include:
More specifically, it may refer to:
Acute bronchitis, caused by viruses or bacteria and lasting several days or weeks Chronic bronchitis, a persistent, productive cough lasting at least three months in two consecutive years.
Chronic bronchitis can be caused by cigarette smoke.
Acute bronchitis is often a symptom of the common cold.
Bronchitis is NOT CONTAGIOUS In bronchitis, the membranes lining the larger air passages (bronchi) become inflamed and an excessive amount of mucus is produced.
The person with bronchitis develops a bad cough to get rid of the mucus.
Sometimes it can lead to pneumonia.
Airflow into and out of the lungs is partly blocked because of the swelling and extra mucus in the bronchi.
Bronchitis is commonly treated with an antibiotic called Amoxicillin or with inhalers as with asthma .
A rescue puffer, Ventolin, Salbutomal or Novo-Salmol, is commonly used for a respiratory emergency (dyspnea).
Other inhalers can be prescribed for daily therapy use.
Chronic bronchitis is one sub-category of COPD (chronic obstructive pulmonary disease or disorder).
Emphysema is another sub-category of COPD. With COPD, each interferes with the absorption of oxygen into the blood stream.
COPD patients can degrade to the point where their breathing system does not recognize high levels of carbon dioxide buildup.
Their breathing mechanism will respond to low levels of oxygen instead.
So applying oxygen for treatment of a COPD patient can potentially shut down the respiratory drive.
However, COPD is a secondary issue.
The oxygen is required for treatment of the main injury or illness.
The main point is to monitor and ensure that if the respiratory drive shuts down, start rescue breathing or CPR (if no signs of circulation).
Symptoms include:
- Coughing up extra mucus, sometimes with blood
- Wheezing Difficulty breathing
- Runny nose
More specifically, it may refer to:
Acute bronchitis, caused by viruses or bacteria and lasting several days or weeks Chronic bronchitis, a persistent, productive cough lasting at least three months in two consecutive years.
Chronic bronchitis can be caused by cigarette smoke.
Acute bronchitis is often a symptom of the common cold.
Bronchitis is NOT CONTAGIOUS In bronchitis, the membranes lining the larger air passages (bronchi) become inflamed and an excessive amount of mucus is produced.
The person with bronchitis develops a bad cough to get rid of the mucus.
Sometimes it can lead to pneumonia.
Airflow into and out of the lungs is partly blocked because of the swelling and extra mucus in the bronchi.
Bronchitis is commonly treated with an antibiotic called Amoxicillin or with inhalers as with asthma .
A rescue puffer, Ventolin, Salbutomal or Novo-Salmol, is commonly used for a respiratory emergency (dyspnea).
Other inhalers can be prescribed for daily therapy use.
Chronic bronchitis is one sub-category of COPD (chronic obstructive pulmonary disease or disorder).
Emphysema is another sub-category of COPD. With COPD, each interferes with the absorption of oxygen into the blood stream.
COPD patients can degrade to the point where their breathing system does not recognize high levels of carbon dioxide buildup.
Their breathing mechanism will respond to low levels of oxygen instead.
So applying oxygen for treatment of a COPD patient can potentially shut down the respiratory drive.
However, COPD is a secondary issue.
The oxygen is required for treatment of the main injury or illness.
The main point is to monitor and ensure that if the respiratory drive shuts down, start rescue breathing or CPR (if no signs of circulation).
Bronchiectasis:
Is a disease that causes localized, irreversible dilatation of part of the bronchial tree.
It is classified as an obstructive lung disease, along with bronchitis and cystic fibrosis.
Involved bronchi are dilated, inflamed, and easily collapsible, resulting in airflow obstruction and impaired clearance of secretions.
Bronchiectasis is associated with a wide range of disorders, but it usually results from necrotizing bacterial infections, such as infections caused by the Staphylococcus or Klebsiella species or Bordetella pertussis.Rene Theophile Hyacinthe Laƫnnec, the man who invented the stethoscope, used his creation to first discover bronchiectasis in 1819.
The disease was researched in greater detail by Sir William Osler in the late 1800s; in fact, it is suspected that Osler actually died of complications from undiagnosed bronchiectasis
It is classified as an obstructive lung disease, along with bronchitis and cystic fibrosis.
Involved bronchi are dilated, inflamed, and easily collapsible, resulting in airflow obstruction and impaired clearance of secretions.
Bronchiectasis is associated with a wide range of disorders, but it usually results from necrotizing bacterial infections, such as infections caused by the Staphylococcus or Klebsiella species or Bordetella pertussis.Rene Theophile Hyacinthe Laƫnnec, the man who invented the stethoscope, used his creation to first discover bronchiectasis in 1819.
The disease was researched in greater detail by Sir William Osler in the late 1800s; in fact, it is suspected that Osler actually died of complications from undiagnosed bronchiectasis
Thursday, September 18, 2008
Medication:
Medication I have been take for several years ago:
The Following Medication is taken in a daily bases:
- Combivent Inhaler ... 2 buffs, 4 times a day.
- Serevent Diskus ... 1 buff 2 time a day.
- Uniphyl tablets ... 1 and 1/2 tablets 900mg in the morning time. (600mg)
- Albuterol Sulf tablets ... 1 tablet twices a day... one in the moring and one in the evening. (4mg)
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Sunday, January 13, 2008
My Daily Health Activities
- Do not sleep earlier than 12:00am at night. (sometime stay up till 3:00am, but lately, after August 2008 when I was admitted to the hospital for pneumonia)
- Wakeup around 8:30am, and like to sleep late in the morning time. (till noon, if there is nothing to worry about)
- Generally when I wakeup in the morning can’t cough out anything from my lungs though I feel it is stuffed with lots of mucus, and lots of wheezing.
- I take my medicine, Uniphyl, and Albuterol tablets.
- Take a Hot bath for around 20 minutes, rarely I cough out little pieces of mucus, with difficulties.
- Take my Inhalers Combivent, and Serevent Diskus around 9:00am
- Start Drink my Coffee (4 measurement cubs a day in the morning) with, maybe donuts, cakes, or anything sweets. That’s when it start coughing and getting the mucus out easily, but not in a large quantities, and the color of the mucus tend to yellow (sometimes dark yellow or light yellow, if I am in a good shape)
- Gradually, when I start moving around and talking to people, the coughing increases and the mucus gets out more easily, bigger chucks, with the color lighter than morning time.
Noon time, and especially after a good lunch, this is the time when I start coughing easily without any effort, with big chucks of mucus and the color of it start clearing out.
Also, at this time (like I say) waking up. I start gaining my strength, and breathing well with no wheezing. - Again, gradually, from the afternoon till I sleep at night, I start functioning better and better in all the activities I do. Less coughing with little and very clear chucks of mucus.
Note: - When I am at home and/or in the weekend, by taking a real good big Lunch and head for an afternoon nap. After I wakeup, I stays in bed, turn/sleep to and on my right side and rise my left hand up, the mucus start coming out real easy. I stay like that for a while until I drain all the mucus out of my lungs.
- Generally when I eat a good meal especially Meats (Beef, Lamb, Chicken, and/or with rice), that’s when I start coughing out mucus, while eating and after I am done eating.
I take my Inhalers Combivent, and Serevent Diskus at 9:00am in the morning and 9:00pm at night. - I take my Combivent Inhaler at 3:00pm afternoon and I wakeup at 3:00am morning/night time, which I like to do this so as to move a little bit and breath normally as an exercise for my lungs.
- I take my Uniphyl (1 and ½), with the Albuterol tablets in the morning time when I wakeup right away.
Note: - Please leave a any comments, if you wish, on anything you like and/or like to write to me.
Tuesday, January 1, 2008
Dr. Visits & Sickness
- 5/6/2009 Wednesday: Dr. Huber, He saw my wife not for me. Everything with her is OK.
- 5/5/2009 Tuesday: Dr. Sheekati, insteaded of Dr. Huber, Major health and pain problem from my month from the Month Alser (not sure!) took two days off from work. I took Amoxicln/clav tab 875mg generic for Augmentin. Till today 5/11/2009 I still have some pain in my right chick side and my tounge is tight from the base of it, and having sharp pain in my thoart to the right side, feels like I am having some sort of flu/cold thing.
- 4/3/2009 Friday: Dr. Wise, she check my nose sinus all the way inside my head, she is everything seems to be ok, no need for anything to do.
- 3/19/2009 Thursday: Dr. Staton, Blood test for thoughdour, X-Ray, and made a CAT Scan for my Head for sinus for Dr. Wise to see 4/3/2009 Friday. Took Lavaquine 500mg. I had mouth alser. The Lavaquine took care of it.
- 1/23/2009 Friday: Dr. H, I was sick, I don't know what was going on. I didn't take the Z (500mg) Antibiotic, because The mucus was very clear, and start getting better and incontrol. Had an X-Ray and it was clear.
- 12/11/2008: Dr. S., Emory, Checkup, Everything was very good.
- 09/23/2008: Dr. Bechara, did the Bronchoscopy procedure and took out couple of stones from the Lungs. (see the Vedio clip I had in this Blog)
- 09/17/2008: Dr. Bechara, I think I visited The Dr. B. before the Bronchoscopy procedure.
- 09/02/08: Dr. S., Visit, after I was released from the Hospital.
- 08/22/2008: Dr. ?!, Did Lungs Bronchoscopy while I was in Emory Hospital
- 08/21/2008 - 08/25/2008: in Emory Hospital, Sick with pneumonia.
- 08/21/2008: Dr. S. Emory, I was very sick and he admin me in the hospital.
- 08/20/2008 - 8/21/2008 3:41am: Gwinnet Medical Center Emergency, Galency Hospital, was very sick, stayed all night, and next day I was in Emory Hospital.
- 08/17/2008: I went to Dr. S. Office and tried to see him, because I missed my appioment in 8/07/2008. I got all the result of the lab/test from 7/17/2008 visit
- 08/07/2008: Dr. S., I don't know if this is the one that I didn't get in for some reason. that why we sat the next appointment for 8/21/2008 and then I was sick with pneumonia.
- 07/17/08: Dr. S., Visit, Did Lots of tests and lab blood test, and x-rays.
- 06/20/2008: Dr. H. Emory Clinic, ?!
- 05/07/2008: Dr. H. Emory Clinic, ?!
- 04/16/2008: Dr. H. Emory Clinic, I was sick with pneumonia and had injection and Antibiotic.
- 03/17/2008: Dr. H. Got the Blood test for the Cholesterol. (see comments)
- 03/14/2008: Dr. H. Emory Clinic, I think I went there for finger problem, he punch it and clean some stuff there.
- 02/01/2008 Dr. H. Emory Clinic, I think I was Sick with pneumonia
- 02/06/2008: Dr. H. Emory Clinic, ?!
- 02/13/2008: Dr. H. Emory Clinic, ?!
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- 1/5/2005: Dr. H . Emory Clinic?!
- 9/1/2004: Dr. H . Emory Clinic?!
- 6/29/2004: Dr. H . Emory Clinic?!
?! Don't remember why.
Dr. S.: Pulmonary/Critical Care Medicine.
See Comments for more details and my notes.
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